*Entamoeba histolytica* is a medically important parasitic protozoan in the genus *Entamoeba* that is transmitted through fecal cysts and is the primary cause of amoebic dysentery.

Entamoeba histolytica

*Acanthamoeba* is a genus of amoeboid protozoans within the supergroup Amoebozoa. It is a human pathogen capable of causing corneal inflammation (keratitis) and blindness.

*Acanthamoeba*

Entamoebas are a subgroup of the eukaryotic supergroup Amoebozoa that exist as active trophozoites and form protective cysts. This subgroup includes the genus *Entamoeba*, which contains commensal or parasitic species.

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Amoebozoa is a eukaryotic supergroup that is divided into slime molds and entamoebas. Slime molds exist in both plasmodial and cellular forms, while entamoebas are characterized by trophozoites that can form cysts.

Amoebozoa

https://openstax.org/details/books/microbiology/

Microbiology covers the scope and sequence requirements for a single-semester microbiology course for non-majors. The book presents the core concepts of microbiology with a focus on applications for careers in allied health. The pedagogical features of the text make the material interesting and accessible while maintaining the career-application focus and scientific rigor inherent in the subject matter. Microbiology’s art program enhances students’ understanding of concepts through clear and effective illustrations, diagrams, and photographs.

Microbiology is produced through a collaborative publishing agreement between OpenStax and the American Society for Microbiology Press. The book aligns with the curriculum guidelines of the American Society for Microbiology.

OpenStax Microbiology Textbook

Slime molds, also known as Eumycetozoa, are a group of organisms in the supergroup Amoebozoa that have historically been classified as animals, fungi, or plants. They are divided into cellular slime molds and plasmodial slime molds.

Slime Molds

Amoeboid movement is a cellular motility process in some eukaryotic microbes, driven by the gel-sol cycling of actin filaments, where fluid soluble actin flows forward to produce temporary membrane extensions called pseudopodia, followed by the remaining cytoplasm flowing up to join the leading edge.

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